CORRECTION BY BENZIMIDAZOL DERIVATIVES OF IMMUNODEFICIENCY AND ABNORMALITIES OF LIPID PEROXIDATION CAUSED BY INTENSE PHYSICAL EXERCISE, PERFORMED AT A LOW ENVIRONMENTAL TEMPERATURE
It has been shown that the usage of benzimidazol actoprotectors in mice leads to a reduction of the depressive impact of stress on the absorption activity of phagocytes and metabolic activity of macrophages. In models of cell and humoral immunity the usage of actoprotectors hindered swimming stress suppression of the immune response in mice. Preventive introduction of tiethasol, bemitil and ethomersol suppressed the activation of lipid peroxidation processes and caused an increase in enzyme antioxidant protection activity in stressed rats.