Endometrial hyperplasia in women of reproductive age at thyroid pathology
The article presents the data on involvement of the thyroid gland disorders in endometrial hyperplasia genesis. Influence of the thyroid gland hypo- and hyperfunction on the hypothalamic-hypophysial-ovarian system is discussed. The issue of correlation between the severity of iodine exchange disorders and the degree of endometrial hyperplasia is clarified. It has been shown that thyroid hormones can indirectly regulate concentration of estrogen and progesterone. Furthermore, production of prolactin and thyroid-stimulating hormone by adenohypophysis in women with abnormal thyroid function is analyzed. We showed that the thyroid gland disorders exert some effects on the function of the adrenal cortex. These data extend our knowledge about the endometrial hyperplasia genesis, thus establishing a scientific basis for new approaches to observation and therapy of women with the thyroid gland pathology.